Medical Health Care Advices

The brains of people suffering from Alzheimer’s disease differ from those of healthy elderly. In these brains are faced with plaques and tangles.

Plaques are deposits of a protein between brain cells. The protein called amyloid. For elderly and particularly in the elderly with Alzheimer’s disease does the degradation of this protein is not good. This causes a kind eiwitbergjes between the brain cells that are likely to transfer messages between the brain cells hamper.

Eventually the nerve cells are also affected. This has been evident in the presence of tangles. A tangle (knot), is a tangle of wire-like proteins in a nerve cell, which the functioning of the nerve cell impossible.

The damage to brain cells is probably because the body reacts with an inflammatory reaction in the presence of plaques. The immune system tries to defuse plaques with toxic substances. That will unfortunately not, but it affects the nerve cells, eventually to. First not function properly. And eventually they die off altogether.

Alzheimer Also created visual spatial disorders which patients get lost and can no longer cover the table. In a later stage creates an Apraxia which a patient all familiar operations such as dressing and undressing and self-sufficiency can not perform. Gradually pull the patient’s back: he is complacent, lose interest in his surroundings, his initiative, and he pays less attention to self. A large percentage of patients may experience mood changes such as depression. Acute deterioration may occur with infection, drug poisoning, hospitalization and major changes such as a sudden move. Known infection with the delirium and hospital admissions. The slow progressive course leading to the degradation of all functions, ultimately leading to a complete dependence on daily care. The average disease duration is eight years.

The development of the disease may be enhanced by isolation. The psychological functions which are used less than the deterioration can go. Spiritual remain in motion stimulates the brain cells that the disease may occur less rapidly

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The disease is characterized by a progressive decline in mental functions. The disease is caused by certain proteins (Amyloid precursor protein : APP) degrade abnormal. This causes abnormal degradation (beta amyloid) which aggregates form. Probably this form of amyloid damaging nerves which then die. In a post mortem examination is greatly reduced cortex (atrophy) found in cells located outside the amyloid deposits (senile plaques), the cell located in amyloid deposits (neuro fibrillary tangles) and deposits around blood vessels. These deposits are not evenly distributed over the cortex. In particular parts of the cortex in which memory functions are localized, are done in the beginning. The disease begins with memory disorders. In the beginning is particularly disturbed imprinting, this new information is not saved. Patient asks, for example several times a day and forget the same arrangements. Information is stored in the past as memories of the past is still available. As the disease progresses, problems arise even in the long term memory. Furthermore arise early in the disease find language disorders such as word problems, disturbances in language comprehension and word production decreased.